

For research and educational purposes only. PepTalk discusses what researchers study in laboratory settings — not medical advice. Products are sold for laboratory research use only.
Research Peptide
GIP/GLP Reta
The lab's leading-edge triple-agonist for metabolic research — freeze-dried, ultra-pure, ready to reconstitute.
GIP/GLP Reta is part of 4 research stacks — 10% off every item plus free bacteriostatic water.

For research and educational purposes only. PepTalk discusses what researchers study in laboratory settings — not medical advice. Products are sold for laboratory research use only.
The short version
Reta is the triple-agonist — a single peptide that switches on three different metabolic receptors at once (GIP, GLP-1, and glucagon). That third pathway is what sets it apart from the dual-agonist compounds before it, and it's why this compound has become one of the most closely watched molecules in current metabolic research.
What researchers are looking at
The body's appetite and energy balance are managed by a handful of gut and pancreatic hormones. Most research compounds in this space mimic one or two of them. The triple-agonist is built to engage three signaling pathways together — GIP, GLP-1, and glucagon — which is the mechanism researchers find most interesting about it.
Two of those pathways (GIP and GLP-1) are studied for how they reduce hunger signaling and improve how the body handles insulin. The third — the glucagon arm — is the novel addition: in published models it's associated with higher energy expenditure and increased fat metabolism in the liver, rather than acting on appetite alone.
Much of the standout data comes from animal and modeling work, where the literature reports unusually rapid changes in body composition. A Phase 2 human trial has since reported body-weight reductions in the range of roughly 24% at the highest concentrations studied — so this is one of the few compounds in the category with genuine human data behind it, though the picture is still early.
GIP pathway
Engages the GIP receptor, studied for its role in insulin action and appetite signaling.
GLP-1 pathway
Activates the GLP-1 receptor, the satiety pathway most familiar from earlier incretin research.
Glucagon pathway
Adds the glucagon arm — associated in published models with higher energy expenditure and hepatic fat metabolism.
Where the evidence stands
an honest mapStriking body-composition changes in animal models
Phase 2 data reported — among the most-advanced in its class
From the lab toward the clinic
Unlike most compounds in early research, the triple-agonist has reached Phase 2 randomized human trials, where the literature reports body-weight reductions of roughly 24% at the highest concentrations studied — a meaningful step from "promising in models" toward rigorous human data.
At a glance
What it is
A triple-agonist peptide
GIP + GLP-1 + glucagon
Studied for
Metabolic research
appetite & energy expenditure
How long it lasts
Long-acting
~140 h half-life
Research stage
In human trials
Phase 2 data reported
How it arrives
Lyophilized powder
Ships freeze-dried for stability and shelf life. Before research use, it's reconstituted with bacteriostatic water.
Bacteriostatic water
Needed to reconstitute
Certificate of analysis
Common questions
All products on FastPeps are labeled Research Use Only (RUO), and this is a legal requirement, not just fine print. It means these compounds are sold strictly for scientific research and laboratory purposes, not for personal use, self-administration, or medical treatment. This label exists because these compounds have not completed the FDA approval process to be sold as drugs or supplements. When you purchase from FastPeps, you are agreeing the product is for research purposes only.
For laboratory and research use only. Sold exclusively for in-vitro research and laboratory experimentation. Not a drug, supplement, or article intended for human or veterinary use; nothing here is medical advice or a claim of therapeutic benefit. Statements describe findings from published research.
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